Lambda λ輕鏈(鼠單克隆抗體) 

廣州健侖生物科技有限公司

此抗體和人免疫球蛋白的λ 輕鏈反應，和κ 輕鏈無交叉反應?？裳芯繋в?lambda; 輕鏈的B 淋巴細胞和漿細胞，細胞外的免疫球蛋白λ 輕鏈也可著色，應注意區(qū)分。研究認為B 淋巴細胞腫瘤的共同特征是只限于單個輕鏈的表達，因此κ 和λ 輕鏈可用于良惡性淋巴病變(反應性淋巴細胞增生和淋巴瘤)的研究。

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Lambda λ輕鏈(鼠單克隆抗體) 

【產(chǎn)品介紹】

細胞定位：細胞漿

克隆號：HP6054

同型：IgG

適用組織：石蠟/冰凍

陽性對照：扁桃體

抗原修復：熱修復（EDTA）

抗體孵育時間：30-60min

Lambda λ輕鏈(鼠單克隆抗體) 

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【公司名稱】 廣州健侖生物科技有限公司
【市場部】     歐

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【騰訊  】 
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-103室

Wright博士說：“在這一點上，利用端粒酶將有毒產(chǎn)物整合進端粒，是非常鼓舞人心的。”
重要的是，和其他許多端粒酶抑制化合物不同，研究人員并沒有在6-thiodG治療小鼠的血液、肝臟和腎臟中觀察到嚴重的副作用。
Shay博士說：“由于端粒酶在幾乎所有的人類腫瘤中表達，這項工作提出了一種潛在創(chuàng)新的方法，靶定端粒酶表達的癌細胞，而對正常細胞的毒副作用較小。我們相信這種小分子將填補未知領(lǐng)域中的一個未曾滿足的癌癥需要，將被迅速應用于臨床。”
我們的機體進化出了一些途徑來擺脫有缺陷的干細胞。來自科羅拉多大學癌癥中心的研究人員在發(fā)表于《干細胞》(Stem Cells)雜志上的一項研究中揭示，其中一條途徑就是通過“重編程”使得被輻射損傷的干細胞分化為不再能夠長久生存的其他細胞。輻射會使得干細胞喪失它的“干性”。這的確有意義：你不會希望受損的干細胞繼續(xù)留在這兒生成受損細胞。
這項研究還證實，當全身遭受輻射時清除輻射損傷干細胞的同一“編程”防衛(wèi)機制導致了血癌生長(延伸閱讀：Sci Rep：福島核輻射后 動物血液出現(xiàn)異常 )。通過重編程這一防衛(wèi)機制，我們或許能夠在全身輻射之后防止癌癥。

This antibody and human immunoglobulin lambda light chain reaction, and kappa light chain no cross-reaction. B lymphocytes and plasma cells with a lambda light chain can be studied. Extracellular immunoglobulin lambda light chains can also be colored, and care should be taken to distinguish them. Studies suggest that the common feature of B-lymphoblastic tumors is limited to the expression of a single light chain, so kappa and lambda light chains can be used in the study of benign and malignant lymphopathies (reactive lymphoproliferation and lymphoma).

Dr. Wright said: "At this point, the use of omerase to integrate toxic products into omeres is very encouraging."
Importantly, and many other different omerase inhibiting compound, the researchers did not treat the blood of mice in the 6-thiodG, liver and kidney were observed serious side effects.
Dr. Shay said: "Because omerase is expressed in almost all human tumors, this work presents a potentially innovative approach, targeting cancer cells express omerase, and toxicity to normal cells smaller. We believe that such small molecules will fill an unmet need for cancer in an unknown area and will be rapidly applied to the clinic. "
Our body has evolved ways to get rid of defective stem cells. Researchers from the University Cancer Center in Colorado in a study published in the (Stem Cells) magazine "stem cell" is disclosed, one of which way is by "reprogramming" such that the radiation damage of stem cells into no longer be able to long-term survival Other cells. Radiation can cause stem cells to lose their "dryness." This does make sense: You do not want damaged stem cells to stay there to create damaged cells.
The study also confirmed that the same clear "programming" defense mechanism of radiation damage stem cells resulted in the growth of blood cancer (Further reading: Sci Rep: After the Fukushima nuclear radiation abnormal animal blood) when subjected to whole body radiation. By reprogramming this defense mechanism, we may be able to prevent cancer after systemic radiation.